Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation
Review
. 2018 Aug;29(8):1181-1188.
doi: 10.1111/jce.13505. Epub 2018 May 9.

Mechanistic subtypes of focal right ventricular tachycardia

Affiliations
Review

Mechanistic subtypes of focal right ventricular tachycardia

Bruce B Lerman et al. J Cardiovasc Electrophysiol. 2018 Aug.

Erratum in

  • Erratum.
    [No authors listed] [No authors listed] J Cardiovasc Electrophysiol. 2019 Jan;30(1):149. doi: 10.1111/jce.13800. Epub 2018 Dec 13. J Cardiovasc Electrophysiol. 2019. PMID: 30618064 No abstract available.

Abstract

Idiopathic sustained focal right ventricular tachycardia (VT) is most frequently due to outflow tract (OT) tachycardia. This arrhythmia is recognized by its characteristic ECG pattern and sensitivity to adenosine. However, there are other forms of idiopathic, focal sustained VT that originate from the right ventricle (RV), which are less well appreciated and easily overlooked. This review will identify the characteristic features and electrophysiologic properties of these forms of RV VT, including those originating from the tricuspid annulus, right ventricular papillary muscles, and moderator band as well as variants of classic RVOT tachycardia and those due to microreentry in the presence of preclinical disease. Recognition of these subtypes of focal RV tachycardia should facilitate targeted therapy.

Keywords: adenosine; catheter ablation; focal tachycardia; ventricular outflow tract; ventricular tachycardia.

PubMed Disclaimer

MeSH terms