Response to the Letter by Poddighe et al. regarding our manuscript "Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis"

Affiliations

¹ Department of Gastroenterology and Hepatology, Kansai Medical University, 2-5-1 Shinmachi, Hirakata, Osaka, 573-1010, Japan.
² Department of Gastroenterology and Hepatology, Kansai Medical University, 2-5-1 Shinmachi, Hirakata, Osaka, 573-1010, Japan. okazaki@hirakata.kmu.ac.jp.

Comment

Kazushige Uchida et al. J Gastroenterol. 2018 Jun.

. 2018 Jun;53(6):793-794.

doi: 10.1007/s00535-018-1460-4.

¹ Department of Gastroenterology and Hepatology, Kansai Medical University, 2-5-1 Shinmachi, Hirakata, Osaka, 573-1010, Japan.
² Department of Gastroenterology and Hepatology, Kansai Medical University, 2-5-1 Shinmachi, Hirakata, Osaka, 573-1010, Japan. okazaki@hirakata.kmu.ac.jp.

No abstract available

Comment on

Basophils activated via TLR signaling may contribute to pathophysiology of type 1 autoimmune pancreatitis.
Yanagawa M, Uchida K, Ando Y, Tomiyama T, Yamaguchi T, Ikeura T, Fukui T, Nishio A, Uemura Y, Miyara T, Okamoto H, Satoi S, Okazaki K. Yanagawa M, et al. J Gastroenterol. 2018 Mar;53(3):449-460. doi: 10.1007/s00535-017-1390-6. Epub 2017 Sep 18. J Gastroenterol. 2018. PMID: 28921377 Free PMC article.
Basophils activated via TLR signaling may contribute to pathophysiology of type I autoimmune pancreatitis".
Poddighe D, Brambilla I, Marseglia GL. Poddighe D, et al. J Gastroenterol. 2018 Jun;53(6):791-792. doi: 10.1007/s00535-018-1456-0. Epub 2018 Apr 16. J Gastroenterol. 2018. PMID: 29663078 No abstract available.