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Comment
. 2018 Jul;102(7):1027-1029.
doi: 10.1097/TP.0000000000002209.

Stop TRPC5 in Kidney Disease: A Game Changer for Renal Allograft Protection?

Xuexiang Wang  1 , Jochen Reiser  1
Affiliations
Comment

Stop TRPC5 in Kidney Disease: A Game Changer for Renal Allograft Protection?

Xuexiang Wang et al. Transplantation. 2018 Jul.
No abstract available

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Conflict of interest statement

The authors declare no funding or conflicts of interest.

Figures

FIGURE 1.
FIGURE 1.
Calciuminvolvement in podocyte function. Scientific historical timeline summarizesmajor publications and findings in the field in the past two decades.
See this image and copyright information in PMC

Comment on

References

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    1. Reiser J, Polu KR, Moller CC. TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function Nat Genet 2005. 37739–744 - PMC - PubMed
    1. Möller CC, Wei C, Altintas MM. Induction of TRPC6 channel in acquired forms of proteinuric kidney disease J Am Soc Nephrol 2007. 1829–36 - PubMed
    1. Dryer SE, Reiser J. TRPC6 channels and their binding partners in podocytes: role in glomerular filtration and pathophysiology Am J Physiol Renal Physiol 2010. 299F689–F701 - PMC - PubMed

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