Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation
Case Reports
. 2018 Apr 14;2(3):114-117.
doi: 10.1016/j.case.2017.12.003. eCollection 2018 Jun.

Intracardiac Thrombus in Leukemia: Role of Cardiac Magnetic Resonance Imaging in Eosinophilic Myocarditis

Affiliations
Case Reports

Intracardiac Thrombus in Leukemia: Role of Cardiac Magnetic Resonance Imaging in Eosinophilic Myocarditis

Charles Allderdice et al. CASE (Phila). .
No abstract available

Keywords: Cardiac magnetic resonance imaging; Eosinophilic myocarditis; Hypereosinophilic syndrome; Intracardiac thrombus.

PubMed Disclaimer

Figures

None
Graphical abstract
Figure 1
Figure 1
Transthoracic echocardiography, (A) apical four-chamber view and (B) apical two-chamber view, showing apical isoechoic density (asterisk) suspicious for left ventricular mural thrombus.
Figure 2
Figure 2
Cardiac magnetic resonance, (A) cine steady-state free precession (SSFP) four-chamber view and (B) cine SSFP three-chamber view, showing a large 2.7-cm apical mass (asterisk) in the setting of normal wall motion and overall preserved left ventricular function and small pericardial effusion. (C) T2-weighted fast spin echo sequences with evidence of hyperintense signal in the apical and inferior myocardial wall adjacent to the thrombus suggestive of myocardial inflammation (blue arrow). (D) Resting first-pass perfusion images did not reveal any gadolinium uptake in the mass, suggesting absence of vascularity. Postgadolinium inversion recovery sequences performed with high inversion times (600 ms) in (E) four-chamber view and (F) two-chamber view revealed lack of delayed enhancement and homogeneously black appearance consistent with thrombus and adjacent apical myocardial inflammation.
Figure 3
Figure 3
Cardiac magnetic resonance, (A) cine steady-state free precession (SSFP) four-chamber view and (B) cine SSFP three-chamber view, showing significant reduction in the size of the apical thrombus (red arrow). Postgadolinium inversion recovery sequences performed in four-chamber view (C,D) revealed no evidence of myocardial fibrosis.

References

    1. Farruggia P., D’Angelo P., Acquaviva A., Trizzino A., Tucci F., Cilloni D. Hypereosinophilic syndrome in childhood: clinical and molecular features of two cases. Pediatr Hematol Oncol. 2009;26:129–135. - PubMed
    1. Rapanotti M., Caruso R., Ammatuna E., Zaza S., Trotta L., Divona M. Molecular characterization of paediatric idiopathic hypereosinophilia. Br J Haematol. 2010;151:440–446. - PubMed
    1. Weller P., Bubley G. The idiopathic hypereosinophilic syndrome. Blood. 1994;83:2759–2779. - PubMed
    1. Ekin S., Sertogullarindan B., Gunbatar H., Arisoy A., Yildiz H. Loeffler’s syndrome: an interesting case report. Clin Respir J. 2016;10:112–114. - PubMed
    1. Miszalski-Jamka T., Szczeklik W., Karwat K., Sokolowska B., Gasior J., Rucinkska M. MRI-based evidence for myocardial involvement in women with hypereosinophilic syndrome. Magn Reson Med Sci. 2015;14:107–114. - PubMed

Publication types

LinkOut - more resources