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Comment
. 2018 Oct 16;115(42):10540-10541.
doi: 10.1073/pnas.1814759115. Epub 2018 Oct 1.

Membrane tethering by the autophagy ATG2A-WIPI4 complex

Martin Graef  1   2
Affiliations
Comment

Membrane tethering by the autophagy ATG2A-WIPI4 complex

Martin Graef. Proc Natl Acad Sci U S A. .
No abstract available

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Conflict of interest statement

The author declares no conflict of interest.

Figures

Fig. 1.
Fig. 1.
The ATG2-Atg18/WIPI complex may tether forming autophagosomes (IM) to the ER and facilitate lipid transport.
See this image and copyright information in PMC

Comment on

References

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    1. Graef M, Friedman JR, Graham C, Babu M, Nunnari J. ER exit sites are physical and functional core autophagosome biogenesis components. Mol Biol Cell. 2013;24:2918–2931. - PMC - PubMed
    1. Böckler S, Westermann B. Mitochondrial ER contacts are crucial for mitophagy in yeast. Dev Cell. 2014;28:450–458. - PubMed
    1. Hamasaki M, et al. Autophagosomes form at ER-mitochondria contact sites. Nature. 2013;495:389–393. - PubMed

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