Correction for Lee et al., Altered ER-mitochondria contact impacts mitochondria calcium homeostasis and contributes to neurodegeneration in vivo in disease models

No authors listed

Published Erratum

No authors listed. Proc Natl Acad Sci U S A. 2018.

. 2018 Oct 16;115(42):E9992.

doi: 10.1073/pnas.1815900115. Epub 2018 Oct 8.

No abstract available

Erratum for

Altered ER-mitochondria contact impacts mitochondria calcium homeostasis and contributes to neurodegeneration in vivo in disease models.
Lee KS, Huh S, Lee S, Wu Z, Kim AK, Kang HY, Lu B. Lee KS, et al. Proc Natl Acad Sci U S A. 2018 Sep 18;115(38):E8844-E8853. doi: 10.1073/pnas.1721136115. Epub 2018 Sep 5. Proc Natl Acad Sci U S A. 2018. PMID: 30185553 Free PMC article.

ER-Mitochondria contact sites: A new regulator of cellular calcium flux comes into play.
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From dysfunctional endoplasmic reticulum-mitochondria coupling to neurodegeneration.
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Mitochondrial Ca²⁺ handling in Huntington's and Alzheimer's diseases - Role of ER-mitochondria crosstalk.
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The entangled ER-mitochondrial axis as a potential therapeutic strategy in neurodegeneration: A tangled duo unchained.
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Mitochondria, calcium, and endoplasmic reticulum stress in Parkinson's disease.
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