Editorial: Ion Channel Trafficking and Cardiac Arrhythmias

Affiliations

¹ Division of Heart and Lungs, Department of Medical Physiology, University Medical Center Utrecht, Utrecht, Netherlands.
² Department of Physiology, University of Kentucky, Lexington, KY, United States.
³ Institute of Biochemistry and Molecular Medicine, Swiss National Centre of Competence in Research TransCure, University of Bern, Bern, Switzerland.

Editorial

Marcel A G van der Heyden et al. Front Physiol. 2018.

. 2018 Sep 25:9:1254.

doi: 10.3389/fphys.2018.01254. eCollection 2018.

¹ Division of Heart and Lungs, Department of Medical Physiology, University Medical Center Utrecht, Utrecht, Netherlands.
² Department of Physiology, University of Kentucky, Lexington, KY, United States.
³ Institute of Biochemistry and Molecular Medicine, Swiss National Centre of Competence in Research TransCure, University of Bern, Bern, Switzerland.

No abstract available

Keywords: Kir2.1; Kv11.1; Nav1.5; TRPM4; autophagy; connexin 43; ion channel trafficking; protein complexes.

Comment on

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1. Basheer W. A., Shaw R. M. (2016). Connexin 43 and Cav1.2 Ion channel trafficking in healthy and diseased myocardium. Circ. Arrhythm. Electrophysiol. 9:e001357. 10.1161/CIRCEP.115.001357 - DOI - PMC - PubMed
1. de Git K. C., de Boer T. P., Vos M. A., van der Heyden M. A. (2013). Cardiac ion channel trafficking defects and drugs. Pharmacol. Ther. 139, 24–31. 10.1016/j.pharmthera.2013.03.008 - DOI - PubMed
1. Li X., Ortega B., Kim B., Welling P. A. (2016). A common signal patch drives AP-1 protein-dependent golgi export of inwardly rectifying potassium channels. J. Biol. Chem. 291, 14963–14972. 10.1074/jbc.M116.729822 - DOI - PMC - PubMed