Pathophysiology of Reperfusion Injury
- PMID: 30485021
- Bookshelf ID: NBK534267
Pathophysiology of Reperfusion Injury
© The Contributors 2011.
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References
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- Allen DG, Xiao XH. Activity of the NA+/H+ exchanger contributes to cardiac damage following ischaemia and reperfusion. Clin Exp Pharmacol Physiol 2000; 27: 727–33. - PubMed
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- Paoni NF, Peale F, Wang F, Errett-baroncini C, Steinmetz H, Toy K,Bai W, Williams PM, Bunting S, Gerritsen ME, Powell-braxton L. Time course of skeletal muscle repair and gene expression following acute hind limb ischemia in mice. Physiol Genomics 2002; 11: 263–72. - PubMed
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- Safronova O,Morita I. Transcriptome remodeling in hypoxic inflammation. J Dent Res 2010; 89: 430–44. - PubMed
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- Hierholzer C, Harbrecht BG, Billiar TR, Tweardy DJ. Hypoxia-inducible factor-1 activation and cyclo-oxygenase-2 induction are early reperfusion-independent inflammatory events in hemorrhagic shock. Arch Orthop Trauma Surg 2001; 121: 219–22. - PubMed
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- Yokoyama K, Kimura M, Nakamura K, Itoman M. Time course of post-ischemic superoxide generation in venous effluent from reperfused rabbit hindlimbs. J Reconstr Microsurg 1999; 15: 215–21. - PubMed
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