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. 2019 Jan;9(1):41-46.
doi: 10.1177/1941874418758902. Epub 2018 Feb 20.

An Unusual Etiology of Acute Spontaneous Intracerebral Hemorrhage

Affiliations

An Unusual Etiology of Acute Spontaneous Intracerebral Hemorrhage

Chitra Sivasankar et al. Neurohospitalist. 2019 Jan.
No abstract available

Keywords: central nervous system infections; cerebrovascular disorders; epilepsy; intracranial hemorrhages; neuropathology; seizures; status epilepticus; techniques.

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Conflict of interest statement

Declaration of Conflicting Interests: The authors declared no potential conflicts of interest with respect to the research, authorship, and/or publication of this article.

Figures

Figure 1.
Figure 1.
Initial noncontrast head computed tomography demonstrating a 2.5 × 3.2 cm right lateral temporal lobe intracerebral hemorrhage.
Figure 2.
Figure 2.
Histopathological findings from right temporal lobe tissue. A, Low-power (×40) hematoxylin–eosin stain of right temporal lobe tissue showing hypercellular parenchyma with lymphoplasmacytic and neutrophilic infiltrate as well as hemorrhage and necrosis (arrows). B, High-power (×500) hematoxylin–eosin stain showing astrocytes with cytopathic effect and intranuclear inclusions (arrows). C, Immunohistochemical staining (×200) showing strong diffuse positivity for herpes simplex virus 1 (HSV-1).
Figure 3.
Figure 3.
Postoperative brain magnetic resonance imaging (MRI) findings. A and B, Postoperative axial MRI of the brain showing diffuse T2 fluid-attenuated inversion recovery signal hyperintensities in the bilateral hemispheres involving cortical and subcortical structures. C and D, Postoperative axial MRI of the brain showing diffuse gyral gadolinium enhancement throughout the right parietal and temporal lobes.
Figure 4.
Figure 4.
A and B, Final noncontrast head computed tomography demonstrating diffuse hypodensities throughout right and left hemisphere with complete effacement of the basal cisterns and uncal herniation.

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