MOTS-c: an equal opportunity insulin sensitizer

Affiliations

¹ Leonard Davis School of Gerontology, University of Southern California, Los Angeles, CA, 90089-0191, USA.
² Leonard Davis School of Gerontology, University of Southern California, Los Angeles, CA, 90089-0191, USA. hassy@usc.edu.

Su-Jeong Kim et al. J Mol Med (Berl). 2019 Apr.

. 2019 Apr;97(4):487-490.

doi: 10.1007/s00109-019-01758-0. Epub 2019 Feb 20.

¹ Leonard Davis School of Gerontology, University of Southern California, Los Angeles, CA, 90089-0191, USA.
² Leonard Davis School of Gerontology, University of Southern California, Los Angeles, CA, 90089-0191, USA. hassy@usc.edu.

No abstract available

Figures

1. Lee C, Zeng J, Drew BG, et al. (2015) The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance. Cell Metab 21:443–454. doi: 10.1016/j.cmet.2015.02.009 - DOI - PMC - PubMed
1. Lee C, Kim KH, Cohen P (2016) MOTS-c: A novel mitochondrial-derived peptide regulating muscle and fat metabolism.Free Radic Biol Med 100:182–187. doi: 10.1016/j.freeradbiomed.2016.05.015 - DOI - PMC - PubMed
1. Lu H, Wei M, Zhai Y et al. (2019) MOTS-c peptide regulates adipose homeostasis to prevent ovariectomy-induced metabolic dysfunction J Mol Med 10.1007/s00109-018-01738-w - DOI - PubMed
1. Szmuilowicz ED, Stuenkel CA, Seely EW (2009) Influence of menopause on diabetes and diabetes risk. Nat Rev Endocrinol 5:553–558. doi: 10.1038/nrendo.2009.166 - DOI - PubMed
1. Paschou SA, Anagnostis P, Pavlou DI, et al. (2018) Diabetes in menopause: risks and management. Curr Vasc Pharmacol 16:1–8. doi: 10.2174/1570161116666180625124405 - DOI - PubMed