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Review
. 2018 Jun 5;11(5):128-134.
doi: 10.1002/cld.704. eCollection 2018 May.

Imaging of abnormal liver function tests

Saivenkat H Vagvala  1 , Stacy D O'Connor  1
Affiliations
Review

Imaging of abnormal liver function tests

Saivenkat H Vagvala et al. Clin Liver Dis (Hoboken). .
No abstract available

PubMed Disclaimer

Figures

Figure 1
Figure 1
Chronic hepatitis C. (A) Ultrasound demonstrating hepatic surface nodularity and a coarsened echotexture, consistent with patient's biopsy‐proven cirrhosis. (B) CT in the same patient shows a hypertrophied left and caudate lobe, liver surface nodularity, and small perigastric varicosities (yellow arrow). SAG, sagittal.
Figure 2
Figure 2
Celiac disease. (A) Small‐bowel follow‐through and (B) CT exhibiting reversal of the jejunoileal mucosal fold pattern, such that the jejunum has a decreased number of folds, whereas the ileum has an increased number of folds.
Figure 3
Figure 3
Alpha‐1‐antitrypsin deficiency. CT demonstrating severe basilar predominant paraseptal/centrilobular emphysema and scarring. Hepatic involvement may result in cirrhosis (not shown).
Figure 4
Figure 4
NAFLD. (A) Ultrasound demonstrates diffuse increased echogenicity and (B) CT demonstrates diffuse decreased attenuation. (C) in‐phase and (D) out‐of‐phase MR imaging exhibits diffuse, homogenous signal loss on out‐of‐phase images. SAG, sagittal.
Figure 5
Figure 5
NASH. Shear wave elastography demonstrating elevated median velocity of 1.8 m/second, consistent with stage F3 fibrosis.
Figure 6
Figure 6
HCC. (A) Arterial‐phase CT shows a 14‐mm enhancing lesion (black open arrow) with capsular enhancement. There is washout on (B) venous phase in this and an adjacent lesion (black arrows).
Figure 7
Figure 7
Sarcoidosis. CT exhibiting hepatomegaly with numerous small hypodense lesions in the liver and spleen.
Figure 8
Figure 8
Hemochromatosis. (A) T2‐weighted MRI demonstrates diffuse low signal, whereas (B) out‐of‐phase and (C) in‐phase MRI exhibit loss of signal on the in‐phase images because of the longer TE.
Figure 9
Figure 9
Budd‐Chiari syndrome. (A) Ultrasound illustrating nonvisualization of the hepatic veins, consistent with chronic occlusion. (B) CT demonstrating inferior vena cava (IVC) narrowing.
Figure 10
Figure 10
Wilson's disease. T2‐weighted MRI exhibiting a shrunken liver with severe parenchymal atrophy and nodular hypointense foci.
Figure 11
Figure 11
Gallstone pancreatitis. (A and B) CT demonstrating a calcified stone (B, red arrow) in the distal common bile duct near the sphincter of Oddi, peripancreatic fluid, and inflammatory stranding (A, green arrows).
Figure 12
Figure 12
Acute cholecystitis. (A) Ultrasound demonstrating a distended gallbladder with wall thickening, dependent sludge, pericholecystic fluid, and hyperemia. (B) CT illustrates gallstones, gallbladder wall thickening, and pericholecystic fluid. (C) Nuclear medicine hepatobiliary scan (cholescintigraphy) shows normal uptake and excretion of radiotracer into the gastrointestinal tract. The gallbladder was not visualized at 2 hours, despite morphine augmentation.
Figure 13
Figure 13
Gallbladder carcinoma. CT of a patient with biopsy‐proven gallbladder carcinoma demonstrates loss of the fat plane between the gallbladder and adjacent liver, and a thickened gallbladder wall with calcifications (black arrows).
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