Pathogenic effector functions of ACPA: Where do we stand?
- PMID: 31005898
- DOI: 10.1136/annrheumdis-2019-215337
Pathogenic effector functions of ACPA: Where do we stand?
Keywords: anti-CCP; arthritis; autoantibodies; autoimmune diseases; osteoclasts.
Conflict of interest statement
Competing interests: None declared.
Comment in
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Response to: "How to communicate in science" by Klareskog et al.Ann Rheum Dis. 2020 Dec;79(12):e165. doi: 10.1136/annrheumdis-2019-216045. Epub 2019 Aug 13. Ann Rheum Dis. 2020. PMID: 31409590 No abstract available.
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How to communicate in science.Ann Rheum Dis. 2020 Dec;79(12):e164. doi: 10.1136/annrheumdis-2019-216016. Epub 2019 Aug 13. Ann Rheum Dis. 2020. PMID: 31409591 No abstract available.
Comment on
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Identification of a novel chemokine-dependent molecular mechanism underlying rheumatoid arthritis-associated autoantibody-mediated bone loss.Ann Rheum Dis. 2016 Apr;75(4):721-9. doi: 10.1136/annrheumdis-2015-208093. Epub 2015 Nov 26. Ann Rheum Dis. 2016. PMID: 26612338 Free PMC article.
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Autoantibodies to citrullinated proteins induce joint pain independent of inflammation via a chemokine-dependent mechanism.Ann Rheum Dis. 2016 Apr;75(4):730-8. doi: 10.1136/annrheumdis-2015-208094. Epub 2015 Nov 27. Ann Rheum Dis. 2016. PMID: 26613766 Free PMC article.
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