Endothelial Cell-Derived Angiocrines Elicit Physiological Cardiomyocyte Hypertrophy

Kazutaka Ueda^{1

2}, Haruhiro Toko^{1

3}, Issei Komuro¹

Affiliations

¹ Department of Cardiovascular Medicine, Graduate School of Medicine (K.U., H.T., I.K.).
² Department of Ubiquitous Preventive Medicine (K.U.).
³ Department of Advanced Translational Research and Medicine in Management of Pulmonary Hypertension, The University of Tokyo, Japan (H.T.).

Editorial

Kazutaka Ueda et al. Circulation. 2019.

. 2019 May 28;139(22):2585-2587.

doi: 10.1161/CIRCULATIONAHA.119.040632. Epub 2019 May 28.

Kazutaka Ueda^{1

2}, Haruhiro Toko^{1

3}, Issei Komuro¹

¹ Department of Cardiovascular Medicine, Graduate School of Medicine (K.U., H.T., I.K.).
² Department of Ubiquitous Preventive Medicine (K.U.).
³ Department of Advanced Translational Research and Medicine in Management of Pulmonary Hypertension, The University of Tokyo, Japan (H.T.).

No abstract available

Keywords: Editorials; endothelium; hypertrophy.

Comment on

Endothelial Cells Regulate Physiological Cardiomyocyte Growth via VEGFR2-Mediated Paracrine Signaling.
Kivelä R, Hemanthakumar KA, Vaparanta K, Robciuc M, Izumiya Y, Kidoya H, Takakura N, Peng X, Sawyer DB, Elenius K, Walsh K, Alitalo K. Kivelä R, et al. Circulation. 2019 May 28;139(22):2570-2584. doi: 10.1161/CIRCULATIONAHA.118.036099. Epub 2019 Mar 29. Circulation. 2019. PMID: 30922063 Free PMC article.