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Editorial
. 2019 Aug;47(8):1161-1163.
doi: 10.1097/CCM.0000000000003872.

Inflammasomes Assemble: Cytoplasmic Guardians or Fallen Heroes?

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Editorial

Inflammasomes Assemble: Cytoplasmic Guardians or Fallen Heroes?

Pratik Sinha et al. Crit Care Med. 2019 Aug.
No abstract available

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References

    1. Martinon F, Burns K, Tschopp J. The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta. Mol Cell 2002; 10: 417–426. - PubMed
    1. Strowig T, Henao-Mejia J, Elinav E, Flavell R. Inflammasomes in health and disease. Nature 2012; 481: 278–286. - PubMed
    1. Howrylak JA, Nakahira K. Inflammasomes: Key Mediators of Lung Immunity. Annu Rev Physiol 2017; 79: 471–494. - PubMed
    1. Wu J, Yan Z, Schwartz DE, Yu J, Malik AB, Hu G. Activation of NLRP3 inflammasome in alveolar macrophages contributes to mechanical stretch-induced lung inflammation and injury. J Immunol 2013; 190: 3590–3599. - PMC - PubMed
    1. Liu T, Zhou Y, Li P, Duan JX, Liu YP, Sun GY, Wan L, Dong L, Fang X, Jiang JX, Guan CX. Blocking triggering receptor expressed on myeloid cells-1 attenuates lipopolysaccharide-induced acute lung injury via inhibiting NLRP3 inflammasome activation. Sci Rep 2016; 6: 39473. - PMC - PubMed

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