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Comment
. 2019 Oct 1;111(16):1234-1236.
doi: 10.1002/bdr2.1573. Epub 2019 Aug 5.

Letter to the Editor

Raymond B Runyan  1 , Ornella I Selmin  2 , Susan M Smith  3 , Jennifer L Freeman  4
Affiliations
Comment

Letter to the Editor

Raymond B Runyan et al. Birth Defects Res. .
No abstract available

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References

REFERENCES

    1. Boyer, A. S., Finch, W. T., & Runyan, R. B. (2000). Trichloroethylene inhibits development of embryonic heart valve precursors in vitro. Toxicol Sci, 53(1), 109-117.
    1. Conolly, R. B., & Lutz, W. K. (2004). Nonmonotonic dose-response relationships: Mechanistic basis, kinetic modeling, and implications for risk assessment. Toxicol Sci, 77(1), 151-157. https://doi.org/10.1093/toxsci/kfh007
    1. Collier, J. M., Selmin, O., Johnson, P. D., & Runyan, R. B. (2003). Trichloroethylene effects on gene expression during cardiac development. Birth Defects Res A Clin Mol Teratol, 67(7), 488-495.
    1. Caldwell, P. T., Thorne, P. A., Johnson, P. D., Boitano, S., Runyan, R. B., & Selmin, O (2008). Trichloroethylene disrupts cardiac gene expression and calcium homeostasis in rat myocytes. Toxicol Sci, 104(1), 135-143.
    1. Caldwell, P. T., Manziello, A., Howard, J., Palbykin, B., Runyan, R. B., & Selmin, O. (2010). Gene expression profiling in the fetal cardiac tissue after folate and low-dose trichloroethylene exposure. Birth Defects Res A Clin Mol Teratol, 88(2), 111-127.

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