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Comment
. 2019 Dec 17;10(6):e02616-19.
doi: 10.1128/mBio.02616-19.

Genomics and Chlamydial Persistence In Vivo

Dan D Rockey  1 , Robert J Suchland  2 , Steven J Carrell  3
Affiliations
Comment

Genomics and Chlamydial Persistence In Vivo

Dan D Rockey et al. mBio. .
No abstract available

Keywords: chlamydia; persistence; trpA.

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Figures

FIG 1
FIG 1
Mutations in trpR (A), trpB (B), and trpA (C) in five persistent strains from patients in sexual health clinics in King County, Washington (patients 1, 2, 3, 6, and 7) discussed in reference and the F/I strain discussed in reference . The figure is formatted similarly to data in reference . Positions in each coding sequence are indicated at the top of each panel (the numbers are centered on the base), and amino acid sequence and changes are indicated below each DNA sequence. Mutant bases and amino acids are indicated by boldface type as in reference .
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References

    1. Somboonna N, Ziklo N, Ferrin TE, Hyuk Suh J, Dean D. 2019. Clinical persistence of Chlamydia trachomatis sexually transmitted strains involves novel mutations in the functional αββα tetramer of the tryptophan synthase operon. mBio 10:e01464-19. doi:10.1128/mBio.01464-19. - DOI - PMC - PubMed
    1. Suchland RJ, Dimond ZE, Putman TE, Rockey DD. 2017. Demonstration of persistent infections and genome stability by whole-genome sequencing of repeat-positive, same-serovar Chlamydia trachomatis collected from the female genital tract. J Infect Dis 215:1657–1665. doi:10.1093/infdis/jix155. - DOI - PMC - PubMed

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