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Case Reports
. 2020 Jul 1;15(7):1041-1043.
doi: 10.2215/CJN.13411119. Epub 2020 May 27.

Secondary Hyperparathyroidism in a Patient with CKD

Affiliations
Case Reports

Secondary Hyperparathyroidism in a Patient with CKD

Ryyan Hyder et al. Clin J Am Soc Nephrol. .
No abstract available

Keywords: CKD; Chronic; Hyperparathyroidism; Renal Insufficiency; Secondary; parathyroid hormone.

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Figures

Figure 1.
Figure 1.
Pathophysiology of secondary hyperparathyroidism. FGF23, fibroblast growth factor-23; PTH, parathyroid hormone.

References

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    1. Cunningham J, Locatelli F, Rodriguez M: Secondary hyperparathyroidism: Pathogenesis, disease progression, and therapeutic options. Clin J Am Soc Nephrol 6: 913–921, 2011. - PubMed
    1. Martin A, David V, Quarles LD: Regulation and function of the FGF23/klotho endocrine pathways. Physiol Rev 92: 131–155, 2012. - PMC - PubMed
    1. Centeno PP, Herberger A, Mun HC, Tu C, Nemeth EF, Chang W, Conigrave AD, Ward DT: Phosphate acts directly on the calcium-sensing receptor to stimulate parathyroid hormone secretion. Nat Commun 10: 4693, 2019. - PMC - PubMed
    1. Ix JH, Isakova T, Larive B, Raphael KL, Raj DS, Cheung AK, Sprague SM, Fried LF, Gassman JJ, Middleton JP, Flessner MF, Block GA, Wolf M: Effects of nicotinamide and lanthanum carbonate on serum phosphate and fibroblast growth factor-23 in CKD: The COMBINE trial. J Am Soc Nephrol 30: 1096–1108, 2019. - PMC - PubMed

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