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Case Reports
. 2021 Feb;48(1):65-67.
doi: 10.1016/j.neurad.2020.06.008. Epub 2020 Jun 27.

COVID-19 as triggering co-factor for cortical cerebral venous thrombosis?

Coline Baudar  1 , Thierry Duprez  2 , Amandine Kassab  1 , Nathalie Miller  3 , Matthieu Pierre Rutgers  4
Affiliations
Case Reports

COVID-19 as triggering co-factor for cortical cerebral venous thrombosis?

Coline Baudar et al. J Neuroradiol. 2021 Feb.
No abstract available

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Figures

Fig. 1
Fig. 1
Images A and D demonstrate parenchymal hyperintensity on FLAIR/T2-weighted views (white arrows), revealing a vasogenic edema in the cortex of the left superior parietal lobule. Diffusion-weighted imaging (DWI) was negative excluding a cytotoxic edema (not shown). Cortical CVT is indicated with red arrows in FLAIR view on D (hyposignal due to deoxyhemoglobin), in pre-contrast T1 weighted view on E (hypersignal due to methemoglobin) and in post-contrast T1-weighted view on F (hyposignal due to the signal intensity switch featuring the so-called “delta sign”). Image B shows 3 hyperintense spots featuring the methemoglobin-containing subacute endoluminal clot (green arrow). Susceptibility-weighted imaging (SWI, image C) reveals the dilatation of the surrounding upstream venous network (white circle).
See this image and copyright information in PMC

References

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