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. 2020 May 4;5(7):1119-1120.

doi: 10.1016/j.ekir.2020.04.023. eCollection 2020 Jul.

The Authors Reply

Shilpa Sharma^{1

2}, Mark R Hanudel³, Joachim H Ix^{4

5}, Isidro B Salusky³, Tomas Ganz^{6

7}, Kim-Lien Nguyen^{2

8}

Affiliations

Affiliations

¹ Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
² Veterans Affairs, Greater Los Angeles Healthcare System, Los Angeles, California, USA.
³ Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
⁴ Division of Nephrology-Hypertension, University of California at San Diego, San Diego, California, USA.
⁵ Veterans Affairs, San Diego Healthcare System, San Diego, California, USA.
⁶ Department of Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
⁷ Department of Pathology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
⁸ Division of Cardiology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.

PMID: 32647775
PMCID: PMC7335947
DOI: 10.1016/j.ekir.2020.04.023

The Authors Reply

Shilpa Sharma et al. Kidney Int Rep. 2020.

. 2020 May 4;5(7):1119-1120.

doi: 10.1016/j.ekir.2020.04.023. eCollection 2020 Jul.

Authors

Shilpa Sharma^{1

2}, Mark R Hanudel³, Joachim H Ix^{4

5}, Isidro B Salusky³, Tomas Ganz^{6

7}, Kim-Lien Nguyen^{2

8}

Affiliations

¹ Division of Nephrology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
² Veterans Affairs, Greater Los Angeles Healthcare System, Los Angeles, California, USA.
³ Department of Pediatrics, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
⁴ Division of Nephrology-Hypertension, University of California at San Diego, San Diego, California, USA.
⁵ Veterans Affairs, San Diego Healthcare System, San Diego, California, USA.
⁶ Department of Medicine, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
⁷ Department of Pathology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.
⁸ Division of Cardiology, David Geffen School of Medicine at UCLA, Los Angeles, California, USA.

PMID: 32647775
PMCID: PMC7335947
DOI: 10.1016/j.ekir.2020.04.023

No abstract available

PubMed Disclaimer

References

1. Milovanova LY, Shvetsov MY, Milovanova SY, et al. Elevated fibroblast growth factor 23 and decreased Klotho levels are associated with diastolic dysfunction in CKD G4–5D patients. Kidney Int Rep. 2020;5:1118. - PMC - PubMed
1. Sharma S., Hanudel M.R., Ix J.H. Elevated fibroblast growth factor 23 levels are associated with greater diastolic dysfunction in ESRD. Kidney Int Rep. 2019;4:1748–1751. - PMC - PubMed
1. Devaraj S., Syed B., Chien A., Jialal I. Validation of an immunoassay for soluble Klotho protein: decreased levels in diabetes and increased levels in chronic kidney disease. Am J Clin Pathol. 2012;137:479–485. - PubMed
1. Seiler S., Rogacev K.S., Roth H.J. Associations of FGF-23 and sKlotho with cardiovascular outcomes among patients with CKD stages 2-4. Clin J Am Soc Nephrol. 2014;9:1049–1058. - PMC - PubMed

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