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Figure 1.. Stress cardiomyopathy during COVID-19 infection.
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
A. Chest x-ray demonstrated diffuse bilateral pulmonary infiltrates. …
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
A. Chest x-ray demonstrated diffuse bilateral pulmonary infiltrates. B. 12-lead ECG acquired within the hour of echocardiogram demonstrated slight ST segment elevations. C. Transthoracic echocardiogram (TTE) demonstrated mid-wall hypokinesis of the left ventricular (LV) with normal contractility of the basal and apical segments, and normal right ventricular systolic function (L-R: 4-chamber, 3-chamber, and 2-chamber images; top panel: end-diastole; bottom: end-systole). D. Short axis TTE images demonstrate normal contractility of LV basal and apical segments and hypokinesis of the mid-ventricular segments (L-R: Basal, mid, and apical images; top panel: end-diastole; bottom: end-systole). Yellow contours in C and D outline the LV endocardial borders.
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
A. Chest x-ray demonstrated diffuse bilateral pulmonary infiltrates. …
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
A. Chest x-ray demonstrated diffuse bilateral pulmonary infiltrates. B. 12-lead ECG acquired within the hour of echocardiogram demonstrated slight ST segment elevations. C. Transthoracic echocardiogram (TTE) demonstrated mid-wall hypokinesis of the left ventricular (LV) with normal contractility of the basal and apical segments, and normal right ventricular systolic function (L-R: 4-chamber, 3-chamber, and 2-chamber images; top panel: end-diastole; bottom: end-systole). D. Short axis TTE images demonstrate normal contractility of LV basal and apical segments and hypokinesis of the mid-ventricular segments (L-R: Basal, mid, and apical images; top panel: end-diastole; bottom: end-systole). Yellow contours in C and D outline the LV endocardial borders.
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
A. Chest x-ray demonstrated diffuse bilateral pulmonary infiltrates. …
Figure 1.. Stress cardiomyopathy during COVID-19 infection.
A. Chest x-ray demonstrated diffuse bilateral pulmonary infiltrates. B. 12-lead ECG acquired within the hour of echocardiogram demonstrated slight ST segment elevations. C. Transthoracic echocardiogram (TTE) demonstrated mid-wall hypokinesis of the left ventricular (LV) with normal contractility of the basal and apical segments, and normal right ventricular systolic function (L-R: 4-chamber, 3-chamber, and 2-chamber images; top panel: end-diastole; bottom: end-systole). D. Short axis TTE images demonstrate normal contractility of LV basal and apical segments and hypokinesis of the mid-ventricular segments (L-R: Basal, mid, and apical images; top panel: end-diastole; bottom: end-systole). Yellow contours in C and D outline the LV endocardial borders.
Figure 2.. Recovery of stress cardiomyopathy during…
Figure 2.. Recovery of stress cardiomyopathy during clinical improvement.
Panels A-C demonstrate data obtained on…
Figure 2.. Recovery of stress cardiomyopathy during clinical improvement.
Panels A-C demonstrate data obtained on the day of extubation, 10 days following data in Figure 1. A. CXR continued to demonstrate pulmonary infiltrates. B. ECG abnormalities had normalized compared to prior. C. TTE demonstrated recovery of LV systolic function with resolution of regional dysfunction (L-R: 4-chamber and short-axis basal, mid, and apical images; top panel: end-diastole; bottom: end-systole). Yellow contours outline the LV endocardial borders.
Figure 2.. Recovery of stress cardiomyopathy during…
Figure 2.. Recovery of stress cardiomyopathy during clinical improvement.
Panels A-C demonstrate data obtained on…
Figure 2.. Recovery of stress cardiomyopathy during clinical improvement.
Panels A-C demonstrate data obtained on the day of extubation, 10 days following data in Figure 1. A. CXR continued to demonstrate pulmonary infiltrates. B. ECG abnormalities had normalized compared to prior. C. TTE demonstrated recovery of LV systolic function with resolution of regional dysfunction (L-R: 4-chamber and short-axis basal, mid, and apical images; top panel: end-diastole; bottom: end-systole). Yellow contours outline the LV endocardial borders.
Hendren NS, Drazner MH, Bozkurt B and Cooper LT Jr. Description and Proposed Management of the Acute COVID-19 Cardiovascular Syndrome. Circulation. 2020. 10.1161/circulationaha.120.047349
-
DOI
-
PMC
-
PubMed
Medina de Chazal H, Del Buono MG, Keyser-Marcus L, Ma L, Moeller FG, Berrocal D and Abbate A. Stress Cardiomyopathy Diagnosis and Treatment: JACC State-of-the-Art Review. J Am Coll Cardiol. 2018;72:1955–1971.
-
PMC
-
PubMed
Matsumori A, Yamada T, Suzuki H, Matoba Y and Sasayama S. Increased circulating cytokines in patients with myocarditis and cardiomyopathy. Br Heart J. 1994;72:561–6.
-
PMC
-
PubMed
Scally C, Abbas H, Ahearn T, Srinivasan J, Mezincescu A, Rudd A, Spath N, Yucel-Finn A, Yuecel R, Oldroyd K, et al. Myocardial and Systemic Inflammation in Acute Stress-Induced (Takotsubo) Cardiomyopathy. Circulation. 2019;139:1581–1592.
-
PMC
-
PubMed