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Review
. 2020 Sep;66(9):101063.
doi: 10.1016/j.disamonth.2020.101063. Epub 2020 Jul 28.

SARS 2 human coronavirus (COVID -19, SARS CoV2)

R B McFee  1
Affiliations
Review

SARS 2 human coronavirus (COVID -19, SARS CoV2)

R B McFee. Dis Mon. 2020 Sep.
No abstract available

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Figures

Fig. 1
Fig. 1
Electron micrograph of SARS-COV2 (COVID −19) From: National Institutes of Health/NIAID.
Figure 1
Figure 1
Scanning electron micrograph of a cell heavily infected with SARS-CoV-2, the virus that causes COVID-19. Image credit: National Institutes for Health (NIH) -NIAID
Figure 2
Figure 2
Strong staining is present in vascular endothelium (arrow) and vascular smooth muscle cells (arrow-head) (11).
Figure 3
Figure 3
COVID-19 Mechanism of cytokine storm in COVID-19 and potential therapy(29). Also potential targets and associated mechanism of action for therapeutic intervention. These will be discussed in detail in the Therapeutics Section.
FIGURE 4
Figure 4
(From Hoffman, et al - 14) SARS and COVID-19 Infectivity/Response Comparisons.
Figure 5
Figure 1
Critically disrupted RAS and Bradykinin pathways COVID-19 BAL samples.(1)
Figure 6
Figure 2
The upregulation of hyaluronan synthases and downregulation of hyaluronidases combined with the BK-induced hyperpermeability of the lung microvasculature leads to the formation of a HA-hydrogel that inhibits gas exchange in the alveoli of COVID-19 patients (1, 16,17).
See this image and copyright information in PMC

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