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Comment

. 2020 Sep;98(3):785.

doi: 10.1016/j.kint.2020.05.018.

The authors reply

David H Ellison¹, Biff F Palmer²

Affiliations

Affiliations

¹ Division of Nephrology and Hypertension, Oregon Health & Science University, Portland, Oregon, USA. Electronic address: ellisond@ohsu.edu.
² Department of Medicine, Division of Nephrology, University of Texas, Southwestern Medical Center, Dallas, Texas, USA.

PMID: 32828241
DOI: 10.1016/j.kint.2020.05.018

Free article

Comment

The authors reply

David H Ellison et al. Kidney Int. 2020 Sep.

Free article

. 2020 Sep;98(3):785.

doi: 10.1016/j.kint.2020.05.018.

Authors

David H Ellison¹, Biff F Palmer²

Affiliations

¹ Division of Nephrology and Hypertension, Oregon Health & Science University, Portland, Oregon, USA. Electronic address: ellisond@ohsu.edu.
² Department of Medicine, Division of Nephrology, University of Texas, Southwestern Medical Center, Dallas, Texas, USA.

PMID: 32828241
DOI: 10.1016/j.kint.2020.05.018

No abstract available

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Comment on

Hyperkalemia in pseudohypoaldosteronism type 2 can be from mutated WNK4, but more often from impaired ubiquitination of normal WNK4.
Healy JK. Healy JK. Kidney Int. 2020 Sep;98(3):784-785. doi: 10.1016/j.kint.2020.02.042. Kidney Int. 2020. PMID: 32828240 No abstract available.

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