CARD9: key player or bystander in cardiac remodeling under hypertension?
- PMID: 32873900
- DOI: 10.1038/s41440-020-00542-9
CARD9: key player or bystander in cardiac remodeling under hypertension?
Comment on
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A potential role of caspase recruitment domain family member 9 (Card9) in transverse aortic constriction-induced cardiac dysfunction, fibrosis, and hypertrophy.Hypertens Res. 2020 Dec;43(12):1375-1384. doi: 10.1038/s41440-020-0507-0. Epub 2020 Jul 9. Hypertens Res. 2020. PMID: 32647279
References
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- Gross O, Gewies A, Finger K, Schafer M, Sparwasser T, Peschel C, et al. Card9 controls a non-TLR signalling pathway for innate anti-fungal immunity. Nature. 2006;442:651–6. - DOI
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- Hsu YM, Zhang Y, You Y, Wang D, Li H, Duramad O, et al. The adaptor protein CARD9 is required for innate immune responses to intracellular pathogens. Nat Immunol. 2007;8:198–205. - DOI
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- Roth S, Ruland J. Caspase recruitment domain-containing protein 9 signaling in innate immunity and inflammation. Trends Immunol. 2013;34:243–50. - DOI
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- Dorhoi A, Desel C, Yeremeev V, Pradl L, Brinkmann V, Mollenkopf HJ, et al. The adaptor molecule CARD9 is essential for tuberculosis control. J Exp Med. 2010;207:777–92. - DOI
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