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Case Reports
. 2020 Nov 23;24(1):658.
doi: 10.1186/s13054-020-03389-1.

SARS-CoV-2-associated encephalitis: arguments for a post-infectious mechanism

Affiliations
Case Reports

SARS-CoV-2-associated encephalitis: arguments for a post-infectious mechanism

Adrien Picod et al. Crit Care. .
No abstract available

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Conflict of interest statement

Authors report no competing interest.

Figures

Fig. 1
Fig. 1
Brain MRI. Initial coronal (a, b) and axial (c, d) fluid-attenuated inversion recovery postcontrast MRI displaying hypersignal or enhancement of meninges, cortical and subcortical regions spread over the insula, the cingula, the medial part of the occipital areas and the internal part of the left temporal lobe. 10-week follow-up MRI (e, f) demonstrates complete disappearance of leptomeningeal and cortical FLAIR hyperintensities and enhancements, except for a subtle left-sided residual nodular leptomeningeal enhancement (arrow) adjacent to a cortical vein
Fig. 2
Fig. 2
Electroencephalographic findings. On admission, the patient’s EEG showed intermittent slow periodic activity predominant on the vertex and left hemisphere (a, 04/20/20), reactive to stimulation. Two weeks later (b, 05/04/20), both EEG and neurological status had nearly fully recovered. Recording features: longitudinal montage (with vertex electrode in a), 10 s per page, 10 µV/mm, 70 Hz high and 0.3 Hz low-frequency filters

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