Inhaled milrinone for sick COVID-19 cohort: A pathophysiology driven hypothesis!
- PMID: 33308938
- PMCID: PMC7700763
- DOI: 10.1016/j.mehy.2020.110441
Inhaled milrinone for sick COVID-19 cohort: A pathophysiology driven hypothesis!
Keywords: Acute respiratory distress syndrome; COVID-19; Endothelial dysfunction; Hypothesis; Inhaled milrinone; Pathophysiology; Systemic inflammation.
Conflict of interest statement
The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.
Comment on
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Inhaled but not intravenous milrinone prevents pulmonary endothelial dysfunction after cardiopulmonary bypass.J Thorac Cardiovasc Surg. 2005 Jul;130(1):83-92. doi: 10.1016/j.jtcvs.2004.09.011. J Thorac Cardiovasc Surg. 2005. PMID: 15999045
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Comparison of inhaled milrinone, nitric oxide and prostacyclin in acute respiratory distress syndrome.World J Crit Care Med. 2017 Feb 4;6(1):74-78. doi: 10.5492/wjccm.v6.i1.74. eCollection 2017 Feb 4. World J Crit Care Med. 2017. PMID: 28224110 Free PMC article.
References
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- Magoon R. Pulmonary vasculature in COVID-19: mechanism to monitoring [ahead of print, 2020 October 5] Korean J Anesthesiol DOI: https://doi.org/10.4097/kja.20536. - PMC - PubMed
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