Tumor-Induced Osteomalacia: Treatment Progress Using Burosumab, an Anti-FGF23 Monoclonal Antibody
- PMID: 33740291
- DOI: 10.1002/jbmr.4280
Tumor-Induced Osteomalacia: Treatment Progress Using Burosumab, an Anti-FGF23 Monoclonal Antibody
Comment on
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Burosumab for the Treatment of Tumor-Induced Osteomalacia.J Bone Miner Res. 2021 Apr;36(4):627-635. doi: 10.1002/jbmr.4233. Epub 2021 Jan 12. J Bone Miner Res. 2021. PMID: 33338281 Free PMC article. Clinical Trial.
References
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- Jan de Beur SM, Miller PD, Weber TJ, et al. Burosumab for the treatment of tumor-induced osteomalacia. J Bone Miner Res. 2021. Epub ahead of print. https://doi.org/10.1002/jbmr.4233.
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- Ovejero D, Lim YH, Boyce AM, et al. Cutaneous skeletal hypophosphatemia syndrome: clinical spectrum, natural history, and treatment. Osteoporos Int. 2016;12:3615-3626.
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- Smith P, Bayliss S, Shinawi M, et al. First report of burosumab (anti-FGF23 monoclonal antibody) for rickets complicating HRAS-associated cutaneous skeletal hypophosphatemia syndrome (abstract). J Bone Miner Res. 2019;34(Suppl 1):75.
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- Mumm S, Huskey M, Cajic A, et al. PHEX 3'-UTR mutation c.*231A>G near the polyadenylation signal is a relatively common, mild, American mutation that masquerades as sporadic or X-linked recessive hypophosphatemic rickets. J Bone Miner Res. 2015;30:137-143.
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- Smith PS, Gottesman GS, Zhang F, et al. X-linked hypophosphatemia: uniquely mild disease associated with PHEX 3'-UTR mutation c.*231A>G (a retrospective, case-control study). J Bone Miner Res. 2020;35:920-931.
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