Hyperventilation as one of the mechanisms of persistent dyspnoea in SARS-CoV-2 survivors
- PMID: 34385265
- PMCID: PMC8361302
- DOI: 10.1183/13993003.01578-2021
Hyperventilation as one of the mechanisms of persistent dyspnoea in SARS-CoV-2 survivors
Abstract
Inadequate exercise hyperventilation should not be overlooked while exploring the causes of exertional dyspnoea in SARS-CoV-2 survivors
Conflict of interest statement
Conflict of interest: E. Vidal-Petiot reports personal fees and non-financial support from Servier, outside the submitted work. A. Cohen-Solal has received grants or honoraria from Novartis, Servier, Daiichi Sankyo, Vifor, Menarini and Cardiorentis, outside of the submitted work. J. Frija-Masson reports non-financial support from Vitalaire, Boehringer Ingelheim, Oxyvie and LVL Medical, outside the submitted work. All other authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Comment in
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Phenotyping long COVID.Eur Respir J. 2021 Aug 26;58(2):2101763. doi: 10.1183/13993003.01763-2021. Print 2021 Aug. Eur Respir J. 2021. PMID: 34244323 Free PMC article.
Comment on
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Deconditioning as main mechanism of impaired exercise response in COVID-19 survivors.Eur Respir J. 2021 Aug 26;58(2):2100870. doi: 10.1183/13993003.00870-2021. Print 2021 Aug. Eur Respir J. 2021. PMID: 33926969 Free PMC article.
References
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- Wasserman KHJ, Sue DY, Casaburi R, et al. Principles of Exercise Testing and Interpretation: Including Pathophysiology and Clinical Applications. 3rd edition.Philadelphia, Lippincott, Williams & Wilkins, 1999.
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