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Published Erratum
. 2021 Nov;35(11):e22020.
doi: 10.1096/fsb2.22020.

corrigendum

No authors listed
Published Erratum

corrigendum

No authors listed. FASEB J. 2021 Nov.
No abstract available

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Figures

FIGURE 2
FIGURE 2
Increased SMC proliferation on uptake of H+C‐ or cocaine‐treated MDM–derived EVs. (A,B) Monomac‐1–derived macrophages were treated with H+C in the presence or absence of GW4869 (1 or 10 μM). Supernatants collected were added on HPASMCs in 1:1 ratio with SMC medium followed by a cell proliferation assay after 2 and 4 d. (C) Uptake of EVs by SMCs. EVs were labeled with fluorescent PKH67 dye (green) and added on HPASMCs (10 μg/24 well) for 2 h. Cells were later washed and fixed, followed by staining with phalloidin (red). (D–F) Exosomes (2 μg) resuspended in PBS, isolated from untreated, cocaine (Coc) treated, HIV‐1 infected and from both HIV‐1‐infected and cocaine‐treated monomac‐1 (D) or primary human monocyte (E) derived macrophages were added to 0.1% serum‐starved HPASMCs overnight followed by replenishing the medium the next day. MTS cell proliferation assay was performed at 48 h after exosome treatment. (F) Representative images of HPASMCs treated with EVs. (G) Western blot analysis of HPASMC total cell extract after 48 h of EV treatment for proliferation marker, PCNA. Data are means ± SD from ≥3 independent experiments. *p < .05, **p < .01, ***p < .001 versus control; # p < .05, ## p < .01, ### p < .001 versus cocaine; $ p < .05, $$$ p < .001 versus HIV
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