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Editorial
. 2021 Oct 25:9:759702.
doi: 10.3389/fcell.2021.759702. eCollection 2021.

Editorial: Ectopic Mineralization of Tissues: Mechanisms, Risk Factors, Diseases, and Prevention

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Editorial

Editorial: Ectopic Mineralization of Tissues: Mechanisms, Risk Factors, Diseases, and Prevention

Hervé Kempf et al. Front Cell Dev Biol. .
No abstract available

Keywords: calcification; chronic disease; mouse model; pathologic; rare disease.

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Conflict of interest statement

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

Figures

Figure 1
Figure 1
Graphical summary of the contents covered in the manuscripts published in the special issue. Ectopic calcification in various “soft” tissues can be associated with both chronic and rare diseases. These diseases may affect one or more modulators of “soft” tissue calcification, which include but are not limited to signaling molecules, receptors, enzymes, and other proteins or inorganic molecules acting as mineralization inhibitors. These activating or inhibiting modulators may in turn alter a variety of cellular, extracellular and systemic parameters in the extracellular matrix causing the initiation and progression of ectopic calcification in various “soft” tissues.

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