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Published Erratum
. 2022 Mar;101(3):652.
doi: 10.1016/j.kint.2022.01.002.

Corrigendum to Balbo BE, Amaral AG, Fonseca JM, et al. Cardiac dysfunction in Pkd1-deficient mice with phenotype rescue by galectin-3 knockout. Kidney Int. 2016;90:580-597

No authors listed
Published Erratum

Corrigendum to Balbo BE, Amaral AG, Fonseca JM, et al. Cardiac dysfunction in Pkd1-deficient mice with phenotype rescue by galectin-3 knockout. Kidney Int. 2016;90:580-597

No authors listed. Kidney Int. 2022 Mar.
No abstract available

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Figures

Figure 8 |
Figure 8 |. Cell proliferation, apoptosis, and fibrosis in renal tissue.
Comparative analyses of renal cell Ki-67 staining among (a) WT (n = 6), HTG+ (n = 10), and HTG– (n = 6) organs, and (d) NC (n = 7), CYG+ (n = 5), and CYG– (n = 7) in whole kidney, and in CYG+ and CYG– in cyst epithelia; comparative analyses of renal TUNEL staining among (b) WT (n = 5), HTG+ (n = 6), and HTG– (n = 5) mice in whole kidney, and in CYG+ and CYG– cyst epithelia, and (e) NC (n = 7), CYG+ (n = 6), and CYG– (n = 6) animals; and comparative analyses of area of interstitial fibrosis among (c) WT (n = 8), HTG+ (n = 11), and HTG– (n = 7) kidneys, and (f) NC (n = 9), CYG+ (n = 7), and CYG– (n = 7) organs. (g) Representative images of Ki-67 staining. (h) Representative images of TUNEL staining. (i) Representative images of Sirius red staining. (g–h) Original magnification, ×400; insets, ×800; bar = 10 μm. (i) Original magnification, ×200; bar = 10 µm. Data were compared by Kruskal–Wallis or Mann–Whitney test, with results expressed as medians (lower to upper quartile). (a–c) **P < 0.01 versus WT; ***P < 0.001 versus WT; †P < 0.05 versus HTG+. (d–f) *P < 0.05 versus NC; ***P < 0.001 versus NC; †P < 0.05 versus CYG+. (d,e) ¥¥ P < 0.01 versus CYG+ in cyst epithelia. CYG+, Pkd1cond/cond:Nestincre; CYG–, Pkd1cond/cond:Nestincre:Lgals3−/−; HTG+, Pkd1+/–; HTG–, Pkd1+/–:Lgals3−/−; NC, Pkd1cond/cond; TUNEL, terminal deoxynucleotidyltransferase–mediated dUTP nick end-labeling; WT, wild type.
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