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Editorial
. 2022 Mar 21;12(3):476.
doi: 10.3390/biom12030476.

Redox Imbalance and Mitochondrial Abnormalities in Kidney Disease

Liang-Jun Yan  1
Affiliations
Editorial

Redox Imbalance and Mitochondrial Abnormalities in Kidney Disease

Liang-Jun Yan. Biomolecules. .

Abstract

The kidneys carry out fundamental life-sustaining functions by removing waste substances, controlling salt and water balance, retaining substances vital to the body such as glucose and proteins, and maintaining blood pH balance [...].

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Conflict of interest statement

The author declares no conflict of interest.

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References

    1. Yan L.J. Folic acid-induced animal model of kidney disease. Anim. Model. Exp. Med. 2021;4:329–342. doi: 10.1002/ame2.12194. - DOI - PMC - PubMed
    1. Yan L.J., Allen D.C. Cadmium-induced kidney injury: Oxidative damage as a unifying mechanism. Biomolecules. 2021;11:1575. doi: 10.3390/biom11111575. - DOI - PMC - PubMed
    1. Yan L.J. Nadh/nad(+) redox imbalance and diabetic kidney disease. Biomolecules. 2021;11:730. doi: 10.3390/biom11050730. - DOI - PMC - PubMed
    1. Zhu Y.-T., Wan C., Lin J.-H., Hammes H.-P., Zhang C. Mitochondrial oxidative stress and cell death in podocytopathies. Biomolecules. 2022;12:403. doi: 10.3390/biom12030403. - DOI - PMC - PubMed
    1. Ito M., Gurumani M.Z., Merscher S., Fornoni A. Glucose- and non-glucose-induced mitochondrial dysfunction in diabetic kidney disease. Biomolecules. 2022;12:351. doi: 10.3390/biom12030351. - DOI - PMC - PubMed