Letter to the editor: Compromised hepatic mitochondrial fatty acid oxidation and reduced markers of mitochondrial turnover in human NAFLD
- PMID: 35491446
- DOI: 10.1002/hep.32551
Letter to the editor: Compromised hepatic mitochondrial fatty acid oxidation and reduced markers of mitochondrial turnover in human NAFLD
Comment on
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Compromised hepatic mitochondrial fatty acid oxidation and reduced markers of mitochondrial turnover in human NAFLD.Hepatology. 2022 Nov;76(5):1452-1465. doi: 10.1002/hep.32324. Epub 2022 Apr 14. Hepatology. 2022. PMID: 35000203 Free PMC article.
References
REFERENCES
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- Moore MP, Cunningham RP, Meers GM, Johnson SA, Wheeler AA, Ganga RR, et al. Compromised hepatic mitochondrial fatty acid oxidation and reduced markers of mitochondrial turnover in human NAFLD. Hepatology. 2022 Jan 9. https://doi.org/10.1002/hep.32324. [Epub ahead of print]
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- Gao M, Qin Y, Li A, Wei S, Liu B, Tian X, et al. Mitoflash generated at the Qo site of mitochondrial Complex III. J Cell Physiol. 2021;236:2920-33.
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- Zhang L, Zhou Y, Wu W, Hou L, Chen H, Zuo BO, et al. Skeletal muscle-specific overexpression of PGC-1α induces fiber-type conversion through enhanced mitochondrial respiration and fatty acid oxidation in mice and pigs. Int J Biol Sci. 2017;13:1152-62.
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- Murphy E, Ardehali H, Balaban RS, DiLisa F, Dorn GW, Kitsis RN, et al. Mitochondrial function, biology, and role in disease: a scientific statement from the American Heart Association. Circ Res. 2016;118:1960-91.
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