Editorial: Complement and COVID-19 Disease
- PMID: 35844521
- PMCID: PMC9284945
- DOI: 10.3389/fimmu.2022.960809
Editorial: Complement and COVID-19 Disease
Keywords: COVID-19; SARS – CoV – 2; complement system; kinin-kallikrein system; thromboinflammation.
Conflict of interest statement
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Comment on
- Editorial on the Research Topic Complement and COVID-19 Disease
References
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- Sidarta-Oliveira D, Jara CP, Ferruzzi AJ, Skaf MS, Velander WH, Araujo EP, et al. . SARS-CoV-2 Receptor is Co-Expressed With Elements of the Kinin-Kallikrein, Renin-Angiotensin and Coagulation Systems in Alveolar Cells. Sci Rep (2020) 10(1):19522. doi: 10.1038/s41598-020-76488-2 - DOI - PMC - PubMed
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- Sodhi CP, Wohlford-Lenane C, Yamaguchi Y, Prindle T, Fulton WB, Wang S, et al. . Attenuation of Pulmonary ACE2 Activity Impairs Inactivation of Des-Arg(9) Bradykinin/BKB1R Axis and Facilitates LPS-Induced Neutrophil Infiltration. Am J Physiol Lung Cell Mol Physiol (2018) 314(1):L17–31. doi: 10.1152/ajplung.00498.2016 - DOI - PMC - PubMed
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