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Editorial
. 2022 Sep;37(5):945-946.
doi: 10.3904/kjim.2022.267. Epub 2022 Sep 1.

The role of neutrophils in the pathogenesis of IPF

Affiliations
Editorial

The role of neutrophils in the pathogenesis of IPF

Yangjin Jegal. Korean J Intern Med. 2022 Sep.
No abstract available

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Conflict of interest statement

No potential conflict of interest relevant to this article was reported.

Comment on

References

    1. Raghu G. Idiopathic pulmonary fibrosis: lessons from clinical trials over the past 25 years. Eur Respir J. 2017;50:1701209. - PubMed
    1. Wynn TA, Vannella KM. Macrophages in tissue repair, regeneration, and fibrosis. Immunity. 2016;44:450–462. - PMC - PubMed
    1. Chua F, Dunsmore SE, Clingen PH, et al. Mice lacking neutrophil elastase are resistant to bleomycin-induced pulmonary fibrosis. Am J Pathol. 2007;170:65–74. - PMC - PubMed
    1. Gregory AD, Kliment CR, Metz HE, et al. Neutrophil elastase promotes myofibroblast differentiation in lung fibrosis. J Leukoc Biol. 2015;98:143–152. - PMC - PubMed
    1. Ishikawa G, Liu A, Herzog EL. Evolving perspectives on innate immune mechanisms of IPF. Front Mol Biosci. 2021;8:676569. - PMC - PubMed

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