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Published Erratum
. 2022 Oct;102(4):940.
doi: 10.1016/j.kint.2022.08.005.

Corrigendum to Zhao Y, Wang C, Hong X, et al. Wnt/β-catenin signaling mediates both heart and kidney injury in type 2 cardiorenal syndrome. Kidney Int. 2019;95:815-829

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Published Erratum

Corrigendum to Zhao Y, Wang C, Hong X, et al. Wnt/β-catenin signaling mediates both heart and kidney injury in type 2 cardiorenal syndrome. Kidney Int. 2019;95:815-829

Yue Zhao et al. Kidney Int. 2022 Oct.
No abstract available

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Figures

Figure 3 |
Figure 3 |. Blockade of Wnt/β-catenin signaling by ICG-001 prevented cardiac injury in mice after transverse aortic constriction (TAC).
(a) Experimental design. The red arrow indicates the timing of TAC surgery, and the black arrow shows the timing of death. (b) Representative Western blots show cardiac protein levels of β–myosin heavy chain (MHC), fibronectin, collagen I, tumor necrosis factor (TNF)–α, active β-catenin, and total β-catenin in various groups as indicated. (c–h) Quantitative data on protein levels of β-MHC, fibronectin, collagen I, TNF-α, active β-catenin, and total β-catenin are presented in given groups as indicated. *P < 0.05 versus sham control (Ctrl) subjects. P < 0.05 versus TAC alone (n = 6). (i) Representative micrographs show histology and β-MHC and fibronectin protein expression in the heart of mice at 8 weeks after TAC. Upper panel, hematoxylin and eosin (HE) staining of heart sections in various groups as indicated; middle panel, immunostaining for cardiac β-MHC; bottom panel, immunostaining for fibronectin in the heart of mice as indicated. Yellow arrows indicate positive staining. Bar = 20 μm. Los, losartan. To optimize viewing of this image, please see the online version of this article at www.kidneyinternational.org.

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