This site needs JavaScript to work properly. Please enable it to take advantage of the complete set of features!

Skip to main page content

Add to Collections

Your saved search

Name of saved search:

Search terms:

Test search terms

Would you like email updates of new search results?

Yes
No

Email: (change)

Frequency:

Which day?

Which day?

Report format:

Send at most:

Send even when there aren't any new results

Optional text in email:

Your RSS Feed

Comment

. 2023 Feb;24(2):85.

doi: 10.1038/s41580-022-00553-z.

Amyloids as kinase signalling platforms

Liming Sun¹

Affiliations

PMID: 36266548
DOI: 10.1038/s41580-022-00553-z

Comment

Amyloids as kinase signalling platforms

Liming Sun. Nat Rev Mol Cell Biol. 2023 Feb.

. 2023 Feb;24(2):85.

doi: 10.1038/s41580-022-00553-z.

Author

Liming Sun¹

Affiliation

¹ Center for Excellence in Molecular Cell Science, Chinese Academy of Sciences, Shanghai, China. liming.sun@sibcb.ac.cn.

PMID: 36266548
DOI: 10.1038/s41580-022-00553-z

No abstract available

PubMed Disclaimer

Comment on

The RIP1/RIP3 necrosome forms a functional amyloid signaling complex required for programmed necrosis.
Li J, McQuade T, Siemer AB, Napetschnig J, Moriwaki K, Hsiao YS, Damko E, Moquin D, Walz T, McDermott A, Chan FK, Wu H. Li J, et al. Cell. 2012 Jul 20;150(2):339-50. doi: 10.1016/j.cell.2012.06.019. Cell. 2012. PMID: 22817896 Free PMC article.

References

Original article

1. Li, J. et al. The RIP1/RIP3 necrosome forms a functional amyloid signaling complex required for programmed necrosis. Cell 150, 339–350 (2012) - DOI

Publication types

Actions

MeSH terms

Actions
Actions

Substances

Actions

LinkOut - more resources

Full Text Sources
- Nature Publishing Group