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Comment
. 2022 Nov;20(11):2488-2490.
doi: 10.1111/jth.15863.

Can KCa3.1 channel activators serve as novel inhibitors of platelet aggregation?

Heike Wulff  1 , Andrew P Braun  2 , Seth L Alper  3
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Free article
Comment

Can KCa3.1 channel activators serve as novel inhibitors of platelet aggregation?

Heike Wulff et al. J Thromb Haemost. 2022 Nov.
Free article
No abstract available

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References

REFERENCES

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    1. Gardos G. The function of calcium in the potassium permeability of human erythrocytes. Biochem Biophys Acta. 1958;30:653-654.
    1. Vandorpe DH, Shmukler BE, Jiang L, et al. cDNA cloning and functional characterization of the mouse Ca2+-gated K+ channel, mIK1. Roles in regulatory volume decrease and erythroid differentiation. J Biol Chem. 1998;273:21542-21553.
    1. Brown BM, Shim H, Christophersen P, Wulff H. Pharmacology of small- and intermediate-conductance calcium-activated potassium channels. Annu Rev Pharmacol Toxicol. 2020;60:219-240.
    1. Ataga KI, Smith WR, De Castro LM, et al. Efficacy and safety of the Gardos channel blocker, senicapoc (ICA-17043), in patients with sickle cell anemia. Blood. 2008;111:3991-3997.

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