LncRNA HOTAIR promotes myocardial fibrosis by suppressing miR-124

Affiliations

¹ Institute of Clinical Medicine, The Second Affiliated Hospital of Hainan Medical University, Haikou 570100, Hainan, PR China.
² Department of Nephrology, The First Affiliated Hospital of Dalian Medical University, Dalian 116044, Liaoning, PR China. Electronic address: shuting970109@163.com.
³ Institute of Clinical Medicine, The Second Affiliated Hospital of Hainan Medical University, Haikou 570100, Hainan, PR China; College of Nursing, Anhui University of Chinese Medicine, Hefei 230012, Anhui, PR China. Electronic address: kr198908@163.com.

Comment

Jian Jiang et al. Int J Cardiol. 2023.

. 2023 Mar 1:374:94.

doi: 10.1016/j.ijcard.2022.12.028. Epub 2022 Dec 15.

¹ Institute of Clinical Medicine, The Second Affiliated Hospital of Hainan Medical University, Haikou 570100, Hainan, PR China.
² Department of Nephrology, The First Affiliated Hospital of Dalian Medical University, Dalian 116044, Liaoning, PR China. Electronic address: shuting970109@163.com.
³ Institute of Clinical Medicine, The Second Affiliated Hospital of Hainan Medical University, Haikou 570100, Hainan, PR China; College of Nursing, Anhui University of Chinese Medicine, Hefei 230012, Anhui, PR China. Electronic address: kr198908@163.com.

No abstract available

Keywords: LncRNA HOTAIR; Myocardial fibrosis; miR-124.

Conflict of interest statement

Declaration of Competing Interest The authors report no relationships that could be construed as a conflict of interest.

LncRNA HOTAIR promotes myocardial fibrosis in atrial fibrillation through binding with PTBP1 to increase the stability of Wnt5a.
Tan W, Wang K, Yang X, Wang K, Wang N, Jiang TB. Tan W, et al. Int J Cardiol. 2022 Dec 15;369:21-28. doi: 10.1016/j.ijcard.2022.06.073. Epub 2022 Jul 3. Int J Cardiol. 2022. PMID: 35787431