Reply to: GOT1 constrains TH17 cell differentiation, while promoting iTreg cell differentiation
- PMID: 36725991
- DOI: 10.1038/s41586-022-05603-2
Reply to: GOT1 constrains TH17 cell differentiation, while promoting iTreg cell differentiation
Comment on
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Metabolic control of TH17 and induced Treg cell balance by an epigenetic mechanism.Nature. 2017 Aug 10;548(7666):228-233. doi: 10.1038/nature23475. Epub 2017 Aug 2. Nature. 2017. PMID: 28783731 Free PMC article.
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GOT1 constrains TH17 cell differentiation, while promoting iTreg cell differentiation.Nature. 2023 Feb;614(7946):E1-E11. doi: 10.1038/s41586-022-05602-3. Epub 2023 Feb 1. Nature. 2023. PMID: 36726001 No abstract available.
References
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- Xu, T. et al. Metabolic control of TH17 and induced Treg cell balance by an epigenetic mechanism. Nature 548, 228–233 (2017). - DOI
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- Xu, W. et al. GOT1 constrains TH17 cell differentiation, while promoting iTreg cell differentiation. Nature, https://doi.org/10.1038/s41586-022-05602-3 (2023).
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- Brolosy, et al. Genetic compensation triggered by mutant mRNA degradation. Nature 568, 193–196 (2019). - DOI
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- Ma, et al. PTC-bearing mRNA elicits a genetic compensation response via Upf3a and COMPASS components. Nature 568, 259–263 (2019). - DOI
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- Wise, D. R. et al. Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction. Proc. Natl Acad. Sci. USA 105, 18782–18787 (2008). - DOI
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