Reply to: "Aberrant GAP43 Gene Expression Is Alzheimer Disease Pathology-Specific"
- PMID: 36897272
- DOI: 10.1002/ana.26639
Reply to: "Aberrant GAP43 Gene Expression Is Alzheimer Disease Pathology-Specific"
Comment on
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Association of Presynaptic Loss with Alzheimer's Disease and Cognitive Decline.Ann Neurol. 2022 Dec;92(6):1001-1015. doi: 10.1002/ana.26492. Epub 2022 Sep 15. Ann Neurol. 2022. PMID: 36056679
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Aberrant GAP43 Gene Expression Is Alzheimer Disease Pathology-Specific.Ann Neurol. 2023 May;93(5):1047-1048. doi: 10.1002/ana.26637. Epub 2023 Mar 21. Ann Neurol. 2023. PMID: 36897291 Free PMC article. No abstract available.
References
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- Lan G, Cai Y, Li A, et al. Association of Presynaptic Loss with Alzheimer's disease and cognitive decline. Ann Neurol 2022;92:1001-1015.
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- Davidsson P, Blennow K, Davidsson SP, Blennow K. Dementia neurochemical dissection of synaptic pathology in Alzheimer's disease. 1998.
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- Bogdanovic N, Davidsson P, Volkmann I, et al. Growth-associated protein GAP-43 in the frontal cortex and in the hippocampus in Alzheimer's disease: an immunohistochemical and quantitative study. J Neural Transm 2000;107:463-478.
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- Spires-Jones TL, Hyman BT. The intersection of amyloid Beta and tau at synapses in Alzheimer's disease. Neuron 2014;82:756-771.
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- Forner S, Baglietto-Vargas D, Martini AC, et al. Synaptic impairment in Alzheimer's disease: a dysregulated symphony. Trends Neurosci 2017;40:347-357.
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