HOTAIR functions as a ceRNA to aggravate myocardial fibrosis through the miR-141/wnt5a axis

Affiliations

¹ The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan City People's Hospital, Qingyuan 511518, Guangdong, China.
² The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan City People's Hospital, Qingyuan 511518, Guangdong, China. Electronic address: xiang888200@126.com.

Comment

Sen Huang et al. Int J Cardiol. 2023.

. 2023 Aug 1:384:74.

doi: 10.1016/j.ijcard.2023.04.030. Epub 2023 Apr 21.

¹ The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan City People's Hospital, Qingyuan 511518, Guangdong, China.
² The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan City People's Hospital, Qingyuan 511518, Guangdong, China. Electronic address: xiang888200@126.com.

No abstract available

Conflict of interest statement

Declaration of Competing Interest The authors report no relationships that could be construed as a conflict of interest.

LncRNA HOTAIR promotes myocardial fibrosis in atrial fibrillation through binding with PTBP1 to increase the stability of Wnt5a.
Tan W, Wang K, Yang X, Wang K, Wang N, Jiang TB. Tan W, et al. Int J Cardiol. 2022 Dec 15;369:21-28. doi: 10.1016/j.ijcard.2022.06.073. Epub 2022 Jul 3. Int J Cardiol. 2022. PMID: 35787431