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Editorial
. 2024 Feb;17(1):102-103.
doi: 10.1007/s12265-023-10435-0. Epub 2023 Sep 5.

FAM3A As a Potential Molecular Target to Regulate Mitochondrial Respiration and ATP Production in Ischemic Heart

Yuxin Hu  1
Affiliations
Editorial

FAM3A As a Potential Molecular Target to Regulate Mitochondrial Respiration and ATP Production in Ischemic Heart

Yuxin Hu. J Cardiovasc Transl Res. 2024 Feb.
No abstract available

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References

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    1. Zhang X, et al. FAM3 gene family: a promising therapeutical target for NAFLD and type 2 diabetes. Metabolism. 2018;81:71–82. - DOI - PubMed
    1. Yan H, et al. FAM3A maintains metabolic homeostasis by interacting with F1-ATP synthase to regulate the activity and assembly of ATP synthase. Metabolism. 2023;139: 155372. - DOI - PubMed
    1. Xu T, et al. FAM3A deficiency - induced mitochondrial dysfunction underlies post-infarct mortality and heart failure. J Cardiovasc Transl Res. 2023. https://doi.org/10.1007/s12265-023-10382-w . - DOI - PubMed - PMC

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