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Comment
. 2023 Sep 12;101(11):500.
doi: 10.1212/WNL.0000000000207784.

Author Response: Clinical Subgroups and Factors Associated With Progression in Patients With Inclusion Body Myositis

Thomas E Lloyd  1 , E Harlan Michelle  1 , Iago Pinal-Fernandez  2 , Andrew L Mammen  2
Affiliations
Comment

Author Response: Clinical Subgroups and Factors Associated With Progression in Patients With Inclusion Body Myositis

Thomas E Lloyd et al. Neurology. .
No abstract available

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References

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    1. Ikenaga C, Date H, Kanagawa M, et al. Muscle transcriptomics shows overexpression of cadherin 1 in inclusion body myositis. Ann Neurol. 2022;91(3):317-328. doi: 10.1002/ana.26304 - DOI - PMC - PubMed
    1. Pinal-Fernandez I, Casal-Dominguez M, Derfoul A, et al. Machine learning algorithms reveal unique gene expression profiles in muscle biopsies from patients with different types of myositis. Ann Rheum Dis. 2020;79(9):1234-1242. doi: 10.1136/annrheumdis-2019-216599 - DOI - PMC - PubMed
    1. Britson KA, Ling JP, Braunstein KE, et al. Loss of TDP-43 function and rimmed vacuoles persist after T cell depletion in a xenograft model of sporadic inclusion body myositis. Sci Transl Med. 2022;14(628):eabi9196. doi: 10.1126/scitranslmed.abi9196 - DOI - PMC - PubMed
    1. Kleefeld F, Uruha A, Schänzer A, et al. Morphologic and molecular patterns of polymyositis with mitochondrial pathology and inclusion body myositis. Neurology. 2022;99(20):e2212-e2222. doi: 10.1212/WNL.0000000000201103 - DOI - PubMed

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