Retracted: TJZYF Improves Endometrial Receptivity through Regulating VEGF and PI3K/AKT Signaling Pathway
- PMID: 38188825
- PMCID: PMC10769636
- DOI: 10.1155/2023/9846512
Retracted: TJZYF Improves Endometrial Receptivity through Regulating VEGF and PI3K/AKT Signaling Pathway
Abstract
[This retracts the article DOI: 10.1155/2022/9212561.].
Copyright © 2023 BioMed Research International.
Retraction of
-
TJZYF Improves Endometrial Receptivity through Regulating VEGF and PI3K/AKT Signaling Pathway.Biomed Res Int. 2022 Sep 23;2022:9212561. doi: 10.1155/2022/9212561. eCollection 2022. Biomed Res Int. 2022. Retraction in: Biomed Res Int. 2023 Dec 29;2023:9846512. doi: 10.1155/2023/9846512. PMID: 36193314 Free PMC article. Retracted.
Similar articles
-
TJZYF Improves Endometrial Receptivity through Regulating VEGF and PI3K/AKT Signaling Pathway.Biomed Res Int. 2022 Sep 23;2022:9212561. doi: 10.1155/2022/9212561. eCollection 2022. Biomed Res Int. 2022. Retraction in: Biomed Res Int. 2023 Dec 29;2023:9846512. doi: 10.1155/2023/9846512. PMID: 36193314 Free PMC article. Retracted.
-
Acupuncture Improves Endometrial Angiogenesis by Activating PI3K/AKT Pathway in a Rat Model with PCOS.Evid Based Complement Alternat Med. 2022 Aug 24;2022:1790041. doi: 10.1155/2022/1790041. eCollection 2022. Evid Based Complement Alternat Med. 2022. PMID: 36062171 Free PMC article.
-
Regulation mechanism of miR-494-3p on endometrial receptivity in mice via PI3K/AKT/mTOR pathway.Gen Physiol Biophys. 2021 Sep;40(5):351-363. doi: 10.4149/gpbˍ2021021. Gen Physiol Biophys. 2021. PMID: 34602449
-
The impact of microRNA-mediated PI3K/AKT signaling on epithelial-mesenchymal transition and cancer stemness in endometrial cancer.J Transl Med. 2014 Aug 21;12:231. doi: 10.1186/s12967-014-0231-0. J Transl Med. 2014. PMID: 25141911 Free PMC article. Review.
-
Activation of PI3K/Akt/mTOR pathway and dual inhibitors of PI3K and mTOR in endometrial cancer.Curr Med Chem. 2014;21(26):3070-80. doi: 10.2174/0929867321666140414095605. Curr Med Chem. 2014. PMID: 24735369 Review.
References
Publication types
LinkOut - more resources
Full Text Sources