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. 2024 Feb 1;19(2):e0298294.
doi: 10.1371/journal.pone.0298294. eCollection 2024.

Expression of Concern: Sustained Oxidative Stress Causes Late Acute Renal Failure via Duplex Regulation on p38 MAPK and Akt Phosphorylation in Severely Burned Rats

PLOS ONE Editors

Expression of Concern: Sustained Oxidative Stress Causes Late Acute Renal Failure via Duplex Regulation on p38 MAPK and Akt Phosphorylation in Severely Burned Rats

PLOS ONE Editors. PLoS One. .
No abstract available

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Figures

Fig 2
Fig 2. Histopathological renal injury in rats at 72 h after burn.
Representative images (magnification = 200 x) showed the histological examination for tubular damage with different treatments postburn using periodic acid–Schiff (PAS) staining. The data indicated burn induced significant renal damage at 72 h, which was improved by inhibiting ROS production or p38 MAPK expression, and deteriorated by inhibiting Akt expression. **P<0.01 vs. Sham; #P<0.05, ##P<0.01 vs. Vehicle. n = 6 rats per group per time point.
See this image and copyright information in PMC

Expression of concern for

References

    1. Feng Y, Liu Y, Wang L, Cai X, Wang D, Wu K, et al.. (2013) Sustained Oxidative Stress Causes Late Acute Renal Failure via Duplex Regulation on p38 MAPK and Akt Phosphorylation in Severely Burned Rats. PLoS ONE 8(1): e54593. doi: 10.1371/journal.pone.0054593 - DOI - PMC - PubMed

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