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Published Erratum

. 2024 Feb 22:15:143.

doi: 10.18632/oncotarget.28524.

Correction: YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model

Tsion Zewdu Minas^#¹, Jenny Han^#¹, Tahereh Javaheri², Sung-Hyeok Hong¹, Michaela Schlederer^{2

3}, Yasemin Saygideğer-Kont¹, Haydar Çelik¹, Kristina M Mueller⁴, Idil Temel¹, Metin Özdemirli⁵, Heinrich Kovar^{6

7}, Hayriye Verda Erkizan¹, Jeffrey Toretsky¹, Lukas Kenner^{2

3

8}, Richard Moriggl^{2

9

4}, Aykut Üren¹

Affiliations

Affiliations

¹ Department of Oncology, Georgetown University Medical Center, Washington, DC, USA.
² Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria.
³ Clinical Institute of Pathology, Medical University of Vienna, Vienna, Austria.
⁴ Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria.
⁵ Department of Pathology, Georgetown University Medical Center, Washington, DC, USA.
⁶ Children's Cancer Research Institute, St. Anna Kinderkrebsforschung, Vienna, Austria.
⁷ Department of Pediatrics, Medical University of Vienna, Vienna, Austria.
⁸ Unit of Pathology of Laboratory Animals, University of Veterinary Medicine, Vienna, Austria.
⁹ Medical University of Vienna, Vienna, Austria.

^# Contributed equally.

PMID: 38386842
PMCID: PMC10883682
DOI: 10.18632/oncotarget.28524

Published Erratum

Correction: YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model

Tsion Zewdu Minas et al. Oncotarget. 2024.

. 2024 Feb 22:15:143.

doi: 10.18632/oncotarget.28524.

Authors

Tsion Zewdu Minas^#¹, Jenny Han^#¹, Tahereh Javaheri², Sung-Hyeok Hong¹, Michaela Schlederer^{2

3}, Yasemin Saygideğer-Kont¹, Haydar Çelik¹, Kristina M Mueller⁴, Idil Temel¹, Metin Özdemirli⁵, Heinrich Kovar^{6

7}, Hayriye Verda Erkizan¹, Jeffrey Toretsky¹, Lukas Kenner^{2

3

8}, Richard Moriggl^{2

9

4}, Aykut Üren¹

Affiliations

¹ Department of Oncology, Georgetown University Medical Center, Washington, DC, USA.
² Ludwig Boltzmann Institute for Cancer Research, Vienna, Austria.
³ Clinical Institute of Pathology, Medical University of Vienna, Vienna, Austria.
⁴ Institute of Animal Breeding and Genetics, University of Veterinary Medicine, Vienna, Austria.
⁵ Department of Pathology, Georgetown University Medical Center, Washington, DC, USA.
⁶ Children's Cancer Research Institute, St. Anna Kinderkrebsforschung, Vienna, Austria.
⁷ Department of Pediatrics, Medical University of Vienna, Vienna, Austria.
⁸ Unit of Pathology of Laboratory Animals, University of Veterinary Medicine, Vienna, Austria.
⁹ Medical University of Vienna, Vienna, Austria.

^# Contributed equally.

PMID: 38386842
PMCID: PMC10883682
DOI: 10.18632/oncotarget.28524

No abstract available

PubMed Disclaimer

Figures

Figure 9. Decreased proliferation was observed in spleens and livers of YK-4-279 treated leukemic mice.
E/F; Mx1-cre mice after two weeks treatment with vehicle (middle row) or YK-4-279 (bottom row) were euthanized and spleen and liver samples were processed for histo-pathology analysis. E/F; control mice that lack cre required for EWS-FLI1 activation served as healthy controls (top row) which did not display any symptoms of disease or any peculiar apoptosis or proliferation. Ki67 was used as a marker of proliferation. Ki67 staining of spleen and liver tissues of leukemic E/F;Mx1-cre mice treated with YK-4-279 show a decreased proliferation compared to vehicle treated mice.

See this image and copyright information in PMC

Erratum for

YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
Minas TZ, Han J, Javaheri T, Hong SH, Schlederer M, Saygideğer-Kont Y, Çelik H, Mueller KM, Temel I, Özdemirli M, Kovar H, Erkizan HV, Toretsky J, Kenner L, Moriggl R, Üren A. Minas TZ, et al. Oncotarget. 2015 Nov 10;6(35):37678-94. doi: 10.18632/oncotarget.5520. Oncotarget. 2015. PMID: 26462019 Free PMC article.

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