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Editorial
. 2024 Mar 28;143(13):1206-1207.
doi: 10.1182/blood.2023023448.

GPR56 in GVL: marker or mechanism?

Affiliations
Editorial

GPR56 in GVL: marker or mechanism?

Audra N Iness et al. Blood. .
No abstract available

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Conflict of interest statement

Conflict-of-interest disclosure: P.B. declares equity in Agenus, Amgen, Johnson & Johnson, Exelixis, and BioNTech and receives research support from Allogene Therapeutics. A.N.I. declares no competing financial interests.

Figures

None
CD8+ T cells derived from patients who remained in CR after alloSCT for AML are characterized by a GPR56+, cytotoxic phenotype in contrast to those derived from patients who later relapsed with AML that exhibit GPR56, immune-suppressive regulatory states. Professional illustration by Patrick Lane, ScEYEnce Studios.

Comment on

References

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