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Published Erratum
. 2024 Jul 16;121(29):e2412548121.
doi: 10.1073/pnas.2412548121. Epub 2024 Jul 8.

Correction for Wang et al., The N-Myc-responsive lncRNA MILIP promotes DNA double-strand break repair through non-homologous end joining

No authors listed
Published Erratum

Correction for Wang et al., The N-Myc-responsive lncRNA MILIP promotes DNA double-strand break repair through non-homologous end joining

No authors listed. Proc Natl Acad Sci U S A. .
No abstract available

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Figures

Fig. 6.
Fig. 6.
MILIP Gapmers inhibit, and in cooperation with CDDP to inhibit, neuroblastoma xenograft growth. (A) MILIP expression in cells with or without transfection of Gapmer.MILIP. (B and C) Introduction of Gapmer.MILIP reduced cell viability (B) and clonogenicity (C). (Scale bar, 1 cm.) (D) Quantitation of relative clonogenicity as shown in C. (E and F) Photographs (E) and growth curves (F) of BE(2)-C xenografts in nu/nu mice treated as indicated (n = 6 mice per group). (Scale bar, 1 cm.) (G) Quantitation of tumor weights as shown in E and F showing that cotreatment with Gapmer.MILIP (10 mg/kg, i.v. injection) and CDDP (1 mg/kg, i.p. injection) induced greater inhibition of BE(2)-C tumor growth than treatment with Gapmer.MILIP or CDDP alone in nu/nu mice (n = 6 mice per group, mean ± SEM, one-way ANOVA followed by Tukey’s multiple comparison test). (H) MILIP expression in representative BE(2)-C tumors (n = 3 tumors per group). (I) Representative microscopic photographs of TUNEL staining on randomly selected tumor tissues from mice treated in E and F (n = 3 tumors per group). (J) Quantitation of TUNEL staining as shown in I (n = 3 tumors per group). IRS: immunoreactive score. Data shown are mean ± SEM (A, B, D, and G) or representative (C) of three independent experiments. One-way ANOVA followed by Tukey’s multiple comparison test.
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