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. 2024 Oct 30;30(4):517-519.
doi: 10.5056/jnm24088.

Opioid-induced Esophageal Dysfunction Masquerading as Type I Achalasia

Affiliations

Opioid-induced Esophageal Dysfunction Masquerading as Type I Achalasia

Andrew Leopold et al. J Neurogastroenterol Motil. .
No abstract available

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Conflict of interest statement

Conflicts of interest: None.

Figures

Figure 1
Figure 1
Barium esophagram, high-resolution esophageal manometry (HREM), and endoluminal functional lumen imaging probe (EndoFLIP) prior to buprenorphine cessation. (A) The barium esophagram shows a narrowing at the gastroesophageal junction with delayed contrast emptying of the esophagus. (B) HREM shows absent esophageal body peristalsis, and a suggestion of elevated integrated relaxation pressure, which could not be quantified due to the inability of the HREM catheter to traverse the lower esophageal sphincter (LES). (C) EndoFLIP demonstrates a low median distensibility index of 0.83 at a maximum diameter of 5.7, with a classification of reduced esophagogastric junction (EGJ) opening and weak contractile response.,
Figure 2
Figure 2
Barium esophagram and high-resolution esophageal manometry (HREM) 1 year after cessation of buprenorphine. (A) The barium esophagram demonstrated normal contrast emptying. (B) The HREM was normal, with integrated relaxation pressure of 15 mmHg (normal ≤ 20 with Diversatek’s ZVU). UES, upper esophageal sphincter; LES, lower esophageal sphincter; EGJ, esophagogastric junction.

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