Extracellular Role for the Intracellular Cell Death Mediator RIPK3 in Myocardial Infarction

Affiliations

¹ Departments of Medicine and Cell Biology; and Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, NY.

^# Contributed equally.

Editorial

Dongze Qin et al. Circulation. 2024.

. 2024 Nov 26;150(22):1812-1814.

doi: 10.1161/CIRCULATIONAHA.124.072172. Epub 2024 Nov 25.

¹ Departments of Medicine and Cell Biology; and Wilf Family Cardiovascular Research Institute, Albert Einstein College of Medicine, Bronx, NY.

^# Contributed equally.

No abstract available

Keywords: CaMKII; DAMP; Editorials; MACE; RIPK3; biomarkers; cell death; inflammation; myocardial infarction; necroptosis.

Conflict of interest statement

None.

Extracellular RIPK3 Acts as a Damage-Associated Molecular Pattern to Exaggerate Cardiac Ischemia/Reperfusion Injury.
Zhang W, Zhang J, Wang Z, Li T, Liu C, Kang X, Cui X, Yang J, Qu H, Duanmu J, Peng Y, Wang K, Jin L, Xie P, Zheng W, Shang H, Liu Y, Tian Z, Liu Z, Jin Y, Li Y, Li N, Zhuo X, Wu Y, Shi X, Ma R, Sun Y, Zhang K, Fang X, Hu X, Dong E, Zhang S, Zhang Y. Zhang W, et al. Circulation. 2024 Nov 26;150(22):1791-1811. doi: 10.1161/CIRCULATIONAHA.123.068595. Epub 2024 Oct 16. Circulation. 2024. PMID: 39411860

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